In the landscape of biological psychiatry, a crucial gap exists – a gap characterised by a lack of insights into the pathophysiological mechanisms that underlie psychiatry symptoms. This gap is not merely a scientific void; it represents a critical need for understanding the intricate interplay between neural activity and symptoms, laying the groundwork for unraveling the neuronal and computational mechanisms that give rise to these symptoms.

To address this pressing gap, my research leverages the diverse methodological toolbox of cognitive and computational neuroscience. This approach not only dissects the fundamental biological mechanisms governing human cognition, but also extends into the clinical arena of mental disorders.